![]() ![]() 1, 3, 4 The SHOCK Trial included cardiac index (CI) of ≤2.2 L/min per m 2 and a pulmonary capillary wedge pressure (PCWP) of ≥15 mm Hg. The SHOCK (Should We Emergently Revascularize Occluded Coronaries for Cardiogenic Shock) and intra‐aortic balloon pump (IABP)‐SHOCK II trials used systolic blood pressure (SBP) measurements of 2.0 mmol/L. 2Ĭontemporary trials and guidelines (Table 1) 3, 4, 5, 6, 7 outline clinical criteria for defining CS and are limited by lack of uniformity. 1 Acute myocardial infarction (MI) accounts for 81% of patient in CS. 1 Clinically this presents as hypotension refractory to volume resuscitation with features of end‐organ hypoperfusion requiring pharmacological or mechanical intervention. ![]() CS is caused by severe impairment of myocardial performance that results in diminished cardiac output, end‐organ hypoperfusion, and hypoxia. Cardiogenic shock (CS) is a common cause of mortality, and management remains challenging despite advances in therapeutic options. ![]()
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